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The number of people with glaucoma worldwide in 2010 and 2020
  1. H A Quigley,
  2. A T Broman
  1. The Glaucoma Service and the Dana Center for Preventive Ophthalmology, Wilmer Ophthalmological Institute, Baltimore, MD, USA
  1. Correspondence to: Harry Quigley Wilmer 122, Johns Hopkins Hospital, 600 N Wolfe Street, Baltimore, MD 21287, USA; hquigley{at}jhmi.edu

Abstract

Aim: To estimate the number of people with open angle (OAG) and angle closure glaucoma (ACG) in 2010 and 2020.

Methods: A review of published data with use of prevalence models. Data from population based studies of age specific prevalence of OAG and ACG that satisfied standard definitions were used to construct prevalence models for OAG and ACG by age, sex, and ethnicity, weighting data proportional to sample size of each study. Models were combined with UN world population projections for 2010 and 2020 to derive the estimated number with glaucoma.

Results: There will be 60.5 million people with OAG and ACG in 2010, increasing to 79.6 million by 2020, and of these, 74% will have OAG. Women will comprise 55% of OAG, 70% of ACG, and 59% of all glaucoma in 2010. Asians will represent 47% of those with glaucoma and 87% of those with ACG. Bilateral blindness will be present in 4.5 million people with OAG and 3.9 million people with ACG in 2010, rising to 5.9 and 5.3 million people in 2020, respectively.

Conclusions: Glaucoma is the second leading cause of blindness worldwide, disproportionately affecting women and Asians.

  • ACG, angle closure glaucoma
  • GEE, generalised estimating equations
  • IOP, intraocular pressure
  • LCL, lower confidence limit
  • OAG, open angle glaucoma
  • UCL, upper confidence limit
  • glaucoma
  • prevalence
  • open angle
  • angle closure
  • ACG, angle closure glaucoma
  • GEE, generalised estimating equations
  • IOP, intraocular pressure
  • LCL, lower confidence limit
  • OAG, open angle glaucoma
  • UCL, upper confidence limit
  • glaucoma
  • prevalence
  • open angle
  • angle closure

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Footnotes

  • Supported in part by PHS Research Grants 02120 (Dr Quigley), 01765 (Core Facility Grant, Wilmer Institute), and the Leonard Wagner Trust, New York.

  • Competing interest: none declared

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