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Otitic hydrocephalus was described by Sir Charles P Symonds upon his observation of intracranial hypertension occurring after otitis media with no clinical or serological evidence of meningeal inflammation.1 In 1936, Ersner and Meyers proposed its pathophysiology to be the obliteration of the lateral cerebral sinus.2 The introduction of antibiotics in the modern era has made the disease and its sequelae a rare phenomenon.3 A case of otitic hydrocephalus in a child, resulting in papilloedema with visual loss, is described.
A 5 year old female presented with pansinusitis, bilateral mastoiditis, and thrombosis of the right lateral sinus demonstrated by magnetic resonance imaging (MRI) and venography (fig 1A). Medical history was insignificant. Cerebral …