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Letter
Authors' response
  1. P Wostyn1,
  2. K Audenaert2,
  3. P P De Deyn3,4
  1. 1Department of Psychiatry, PC Sint-Amandus, Beernem, Belgium
  2. 2Department of Psychiatry, Ghent University Hospital, Ghent, Belgium
  3. 3Department of Neurology and Memory Clinic, Middelheim General Hospital (ZNA), Antwerp, Belgium
  4. 4Laboratory of Neurochemistry and Behavior, Institute Born-Bunge, University of Antwerp, Antwerp, Belgium
  1. Correspondence to Dr Peter Wostyn, Department of Psychiatry, PC Sint-Amandus, Reigerlostraat 10, 8730 Beernem, Belgium; wostyn.peter{at}skynet.be

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Thank you for giving us the opportunity to respond to the letter from Professor Emeritus Phillips concerning our recent paper titled “Alzheimer's disease and glaucoma: is there a causal relationship?”.1 In this article, we presented evidence supporting the hypothesis that there could be a causal relationship between Alzheimer's disease (AD) and glaucoma that might be explained by decreased cerebrospinal fluid pressure (CSFP) in patients with AD. Evidence for this hypothesis was gained from CSFP measurement studies in patients with AD and patients with glaucoma. Opposing our hypothesis, Professor Emeritus Phillips cites his 1996 study, published in Acta Ophthalmologica Scandinavica, which rejected the hypothesis that normal-tension glaucoma (NTG) might be due to low CSFP.2 In that study, three patients who had optic neurectomies for meningiomas showed only pale optic discs without evidence …

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