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Torpedo Maculopathy
  1. Pamela R Golchet1,
  2. Lee M Jampol1,
  3. Jeevan R Mathura, Jr.2,
  4. Mark J Daily3,*
  1. 1 Department of Ophthalmology, Northwestern University Feinberg School of Medicine, United States;
  2. 2 2Department of Ophthalmology, The George Washington University, Washington, DC, United States;
  3. 3 Department of Ophthalmology, Loyola University Medical School, Maywood, IL, United States
  1. Correspondence to: Mark Daily, Loyola University Medical School, 343 East Prairie Avenue, Wheaton, Illinois, 60187, United States; markjdaily{at}aol.com

Abstract

Aim: To describe the fluorescein angiographic, fundus autofluorescence, and optical coherence tomography (OCT) findings in patients with a unique unilateral lesion of the temporal macula previously named torpedo maculopathy.

Method: This study was a retrospective, observational case series. The medical records of thirteen patients, age 1 to 68 years, seen between 1982 to 2009 were reviewed. Patients were evaluated for lesion features and course on follow-up, visual acuity, fluorescein angiography, visual field defects, fundus autofluorescence, and OCT findings.

Results: In all thirteen patients, the lesion was flat, torpedo shaped, solitary and involved the temporal macula. The hypopigmented lesion had well-defined margins and a characteristic leading edge which pointed toward the center of the macula. Fluorescein angiography revealed transmission hyperfluorescence of the lesion. OCT indicated a thin abnormal retinal pigment epithelium (RPE) signal and Humphrey Visual Field (HVF) testing revealed a corresponding blind spot. Fundus autofluorescence performed on one patient was dark in the affected area.

Conclusion: Torpedo maculopathy is an apparently congenital hypopigmented torpedo-shaped lesion of the temporal macula. Although it may result in a corresponding visual field defect, these non-foveal lesions do not affect central visual acuity.

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