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I would like to thank Silveira et al for their article, ‘Toxoplasmosis gondii in the peripheral blood of patients with acute and chronic toxoplasmosis’.1 It adds new insight to the pathogenesis of the reactivation of ocular toxoplasmosis, which is known to develop at the border of old scars and is attributed to the rupture of tissue cysts located within the old lesions. But sometimes new lesions are found to occur at a distance from the old scar. The exact mechanism of these findings of reactivation in ocular toxoplasmic patients is not known. The authors studied 20 patients including acute infected patients, patients with recurrent active toxoplasmic retinochoroiditis, patients with old toxoplasmic retinal scars …
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