Patterns of Early Visual Field Loss in Open-Angle Glaucoma
References (10)
- et al.
Comparison of visual field defects in the low-tension glaucomas with those in the high-tension glaucomas
Am. J. Ophthalmol.
(1984) - et al.
Comparison of visual field defects in normal-tension glaucoma and high-tension glaucoma
Am. J. Ophthalmol.
(1986) Low-tension glaucoma. A critical review and new material
Surv. Ophthalmol.
(1980)- et al.
A comparative study of visual fields of patients with low-tension glaucoma and those with chronic simple glaucoma
- et al.
Comparison of glaucomatous visual field defects in patients with high and with low intraocular pressures
Cited by (78)
Overall topographic frequency of glaucoma defects. in memory of Erich Seidel (1882-1948)
2011, Archivos de la Sociedad Espanola de OftalmologiaComparison of Optic Nerve Head Topography and Visual Field in Eyes with Open-angle and Angle-closure Glaucoma
2008, OphthalmologyCitation Excerpt :Whether the more diffuse nature of PACG field loss results from a different exposure to IOP in height and duration or relates to a difference in pathophysiology is not clear. There is evidence that eyes damaged at lower IOP in POAG have more localized field defects than POAG cases with higher IOP levels,29,30 although other authors found that this association was not strong.31 It is tempting to speculate that any differences between eyes with PACG and POAG may relate to differences in their optic disc structure or the susceptibility of retinal ganglion cells to injury.
Alterations in the choroid in hypercholesterolemic rabbits: Reversibility after normalization of cholesterol levels
2007, Experimental Eye ResearchCitation Excerpt :However, few studies have examined ultrastructural changes in choroidal and retinal vascularization during hyperlipidemia, despite epidemiological (Klein et al., 2000; Wong et al., 2001, 2002a,b) and clinical evidences of alterations in patients with this condition. Hyperlipidemic patients are found to have a generalized decrease of retinal sensitivity in the visual field (Hayreh, 1970, 1971; Hayreh et al., 1970; Greve and Geijssen, 1983; Phelps et al., 1983; Caprioli et al., 1987; Schulzer et al., 1990; Terrés et al., 1992) and/or areas of peripapillary atrophy on fundus examination (Jonas et al., 1990, 1992; Tuulonen and Airaksinen, 1992) and histopathology (Kubota et al., 1996). These findings are probably related to choroidal ischemia and alteration of the retinal pigment epithelium (RPE)–photoreceptor complex.
This study was supported in part by grants from Research to Prevent Blindness, Inc., the Connecticut Lion's Eye Research Foundation, and Foresight, Inc. Dr. Miller was supported by training grant 5 T32 EY07000-10 from the National Eye Institute.
Reprint requests to Joseph Caprioli, M.D., Yale University School of Medicine, P.O. Box 3333, New Haven, CT 06510-8061.