Gelatinous drop-like corneal dystrophy (GDLD) is a rare autosomal recessive disorder that is most often seen in Japan. This bilateral dystrophy usually presents in the first decade of life and is associated with a decrease of visual acuity. Typically, a mulberry-like opacity is present with protuberant subepithelial mounds that grow with age. Corneal neovascularisation (NV) also accompanies advanced cases.¹ Corneal transplantation is the major therapeutic option for GDLD, but because NV can significantly increase the risk of graft rejection, a better understanding of the mechanism(s) for the corneal NV would be valuable.