Preliminary CommunicationsCLINICAL SIGN OF OBSTRUCTED AXOPLASMIC TRANSPORT
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Evidence for an enduring ischaemic penumbra following central retinal artery occlusion, with implications for fibrinolytic therapy
2015, Progress in Retinal and Eye ResearchCitation Excerpt :Of note, many CWSs are described as being “isolated” when, in fact, they are sentinels of the boundary(s) of ischaemic infarction; this mistaken impression arises because necrotic inner retina is absorbed much more quickly than the axoplasmic debris accumulating along its borders (McLeod, 1981, 2005). On occasion, axoplasmic debris will accumulate within an area of infarction instead of marking the ischaemic boundary (Fig. 2), albeit the embedded white material is always located close to that boundary (McLeod, 1975, 2009). This implies that the deeper part of the inner retina (i.e. the middle retina comprising the inner plexiform layer [IPL], INL and OPL) may be more extensively infarcted than the superficial inner retina (i.e. the GCL and NFL).
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