Abstract
Purpose:
To evaluate the mechanism of formation of the transient shallow anterior chamber in Vogt-Koyanagi-Harada syndrome.
Methods:
Two patients with Vogt-Koyanagi-Harada syndrome with shallow anterior chambers were examined with an ultrasound biomicroscope.
Results:
A ciliochoroidal detachment, which was not obvious on ophthalmoscopic examination, was clearly demonstrated in both patients by ultra-sound biomicroscopy. The detachment disappeared after systemic corticosteroid therapy.
Conclusion:
The shallowing of the anterior chamber in two patients with Vogt-Koyanagi-Harada syndrome was caused by suprachoroidal effusion secondary to inflammation of the uvea.
MeSH terms
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Anterior Chamber / diagnostic imaging*
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Anterior Chamber / pathology
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Anterior Eye Segment / diagnostic imaging
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Anterior Eye Segment / pathology
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Anti-Inflammatory Agents / therapeutic use
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Choroid Diseases / diagnostic imaging
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Choroid Diseases / etiology
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Ciliary Body / diagnostic imaging
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Ciliary Body / pathology
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Dexamethasone / therapeutic use
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Female
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Humans
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Microscopy
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Middle Aged
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Ultrasonography
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Uveal Diseases / diagnostic imaging
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Uveal Diseases / etiology
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Uveomeningoencephalitic Syndrome / complications
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Uveomeningoencephalitic Syndrome / diagnostic imaging*
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Uveomeningoencephalitic Syndrome / drug therapy
Substances
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Anti-Inflammatory Agents
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Dexamethasone